Unilateral periodic limb movements during sleep in corticobasal degeneration
Identifieur interne : 004656 ( Main/Exploration ); précédent : 004655; suivant : 004657Unilateral periodic limb movements during sleep in corticobasal degeneration
Auteurs : Jorge Iriarte [Espagne] ; Manuel Alegre [Espagne] ; Javier Arbizu [Espagne] ; Purificaci N De Castro [Espagne]Source :
- Movement Disorders [ 0885-3185 ] ; 2001-11.
Descripteurs français
- Pascal (Inist)
- Wicri :
- topic : Homme.
English descriptors
- KwdEn :
- Aged, Basal Ganglia Diseases (complications), Basal Ganglia Diseases (diagnosis), Basal Ganglia Diseases (pathology), Basal Ganglia Diseases (physiopathology), Basal ganglion, Brain (pathology), Brain (radionuclide imaging), Case study, Degeneration, Dominance, Cerebral, Electrodiagnosis, Emission tomography, Human, Humans, Involuntary movement, Limb, Male, Neurodegenerative Diseases (complications), Neurodegenerative Diseases (diagnosis), Neurodegenerative Diseases (pathology), Neurodegenerative Diseases (physiopathology), Nocturnal Myoclonus Syndrome (etiology), Nocturnal Myoclonus Syndrome (physiopathology), Pathogenesis, Polygraphy, Polysomnography (methods), Positron, Sleep, Sleep Initiation and Maintenance Disorders (etiology), Sleep disorder, Tomography, Emission-Computed, Unilateral, Video recording.
- MESH :
- complications : Basal Ganglia Diseases, Neurodegenerative Diseases.
- diagnosis : Basal Ganglia Diseases, Neurodegenerative Diseases.
- etiology : Nocturnal Myoclonus Syndrome, Sleep Initiation and Maintenance Disorders.
- methods : Polysomnography.
- pathology : Basal Ganglia Diseases, Brain, Neurodegenerative Diseases.
- physiopathology : Basal Ganglia Diseases, Neurodegenerative Diseases, Nocturnal Myoclonus Syndrome.
- radionuclide imaging : Brain.
- Aged, Dominance, Cerebral, Humans, Male, Tomography, Emission-Computed.
Abstract
Periodic limb movements in sleep (PLM) have been associated with several degenerative disorders and other focal or diffuse diseases. We present a patient with clinical diagnosis of corticobasal degeneration and sleep complaints, in whom video recording and polygraphic study confirmed the presence of right PLM. The unilaterality of the movements and the positron emission tomography findings (hypometabolism in the left frontoparietal, basal ganglia, and thalamic areas) suggest that the loss of inhibitory descending central pathways, with origin in the cortex or basal ganglia, may be involved in the pathogenesis of PLM. © 2001 Movement Disorder Society.
Url:
DOI: 10.1002/mds.1227
Affiliations:
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Le document en format XML
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<term>Basal Ganglia Diseases (diagnosis)</term>
<term>Basal Ganglia Diseases (pathology)</term>
<term>Basal Ganglia Diseases (physiopathology)</term>
<term>Basal ganglion</term>
<term>Brain (pathology)</term>
<term>Brain (radionuclide imaging)</term>
<term>Case study</term>
<term>Degeneration</term>
<term>Dominance, Cerebral</term>
<term>Electrodiagnosis</term>
<term>Emission tomography</term>
<term>Human</term>
<term>Humans</term>
<term>Involuntary movement</term>
<term>Limb</term>
<term>Male</term>
<term>Neurodegenerative Diseases (complications)</term>
<term>Neurodegenerative Diseases (diagnosis)</term>
<term>Neurodegenerative Diseases (pathology)</term>
<term>Neurodegenerative Diseases (physiopathology)</term>
<term>Nocturnal Myoclonus Syndrome (etiology)</term>
<term>Nocturnal Myoclonus Syndrome (physiopathology)</term>
<term>Pathogenesis</term>
<term>Polygraphy</term>
<term>Polysomnography (methods)</term>
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<term>Sleep</term>
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<term>Sleep disorder</term>
<term>Tomography, Emission-Computed</term>
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<term>Neurodegenerative Diseases</term>
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<term>Brain</term>
<term>Neurodegenerative Diseases</term>
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<term>Nocturnal Myoclonus Syndrome</term>
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<term>Electrodiagnostic</term>
<term>Enregistrement vidéo</term>
<term>Etude cas</term>
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<term>Membre</term>
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<front><div type="abstract" xml:lang="en">Periodic limb movements in sleep (PLM) have been associated with several degenerative disorders and other focal or diffuse diseases. We present a patient with clinical diagnosis of corticobasal degeneration and sleep complaints, in whom video recording and polygraphic study confirmed the presence of right PLM. The unilaterality of the movements and the positron emission tomography findings (hypometabolism in the left frontoparietal, basal ganglia, and thalamic areas) suggest that the loss of inhibitory descending central pathways, with origin in the cortex or basal ganglia, may be involved in the pathogenesis of PLM. © 2001 Movement Disorder Society.</div>
</front>
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